>>35824080First, if you're interested in how the pharmacokinetics and pharmacodynamics of endogenously produced androgens differ from, for example, Decapeptyl SR + some testosterone depot I suggest you consult a textbook on gonadal steroid physiology. This isn't an accepted model of the normal endocrine response and it does not even attempt to mimic it. Your abuse of data you don't understand is not my problem.
Second, changes specific to diet were explained in my last post. The point is that there's a normal relationship between androgen secretion and androgen receptor sensitivity that is preserved with varying dietary fat load (and, if anything, diminished from the FFA-induced anabolic resistance, not increased). To understand whether there is a net gain in androgen receptor signaling, what you would need to do is suppress both androgen receptor resistance and homeostatic testosteronemia simultaneously. The gold-standard treatment for hypogonadism or "borderline hypogonadism" is pharmacologic elevation and not a slab of butter for precisely this reason - it is by definition (like any other drug therapy) throwing the patient out of homeostatic balance - and incidentally a lot of the side effects and nuances of treatment derive from the system trying to restore it.